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Manganese compounds and fume

Airborne particulate or fume from manganese compounds during occupational processing.

Airborne form

fume

The form describes what is suspended in air at the point of exposure. A material can produce more than one form, such as dust during handling and fume during heating.

Health effects

manganismweaknessinsomniamental confusionmetal fume fevercoughchest tightnessbreathing difficultyvomitingkidney damage

Evidence

The current NIOSH Pocket Guide identifies manganese compounds and fume as an airborne form with inhalation and ingestion routes and reports manganism, metal fume fever, cough, chest tightness, breathing difficulty, and kidney damage among effects.

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Exposure Limits; Exposure Routes; Symptoms; Target Organs · 1
Exposure Routes inhalation, ingestion. Symptoms Manganism ... metal fume fever: dry throat, cough, chest tightness, dyspnea ... vomiting ... kidney damage. Target Organs respiratory system, central nervous system, blood, kidneys.

NIOSH lists inhalation and ingestion routes for manganese compounds and fume and reports manganism, asthenia, insomnia, mental confusion, metal fume fever, dry throat, cough, chest tightness, breathing difficulty, vomiting, lassitude, and kidney damage; target organs include the respiratory system, central nervous system, blood, and kidneys.

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Symptoms and target organs · 1
Exposure Routes: inhalation, ingestion. Symptoms: Manganism; asthenia, insomnia, mental confusion; metal fume fever: dry throat, cough, chest tightness, dyspnea (breathing difficulty), rales, flu-like fever; low-back pain; vomiting; malaise; lassitude; kidney damage. Target Organs: respiratory system, central nervous system, blood, kidneys.

A NIOSH HHE investigated manganese-dust exposure in dry-cell battery manufacturing after a report of suspected Parkinson-like neurological effects and evaluated respiratory-protection performance and symptoms associated with overexposure.

Source: pdf ↗ · saved source
page 3 · Introduction · 1
In December 1990, the National Institute for Occupational Safety and Health (NIOSH) received a report from an occupational physician of suspected Parkinson-like neurological effects as a result of manganese (Mn) exposure in the powder areas of the Duracell Battery Company in Cleveland, Tennessee. On March 13-14, 1991, NIOSH investigators conducted the initial site visit of the Health Hazard Evaluation (HHE) at this non-unionized dry cell battery manufacturing plant. The objectives of the HHE were to estimate the workers’ exposures to Mn dust, to evaluate the effectiveness of the respiratory protection program, and to screen the workers for any symptoms associated with Mn overexposure.

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